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A look at bed sores, pressure sores |
Pressure Sores:
- AKA decubitus ulcers, bedsores are preventable
- Caused by constant deficiency of blood supply to tissues
- Typically over bony prominences (sacrum, ischial tub. heel, great. troch.)
- Pressure causes ischemia, which causes necrosis
- Necrotic tissue can lead to infections, this can prevent normal scar tissue from forming
- Infection is usually localized and self limiting
- Proteolytic enzymes from bacteria and macrophages dissolve necrotic tissue and cause a foul smelling discharge
- Painful, fever, increased white blood cell count
- If ulceration is large, pain and toxicity lead to loss of appetite, renal insufficiency, debilitation
- The progression is as follows; Hyperemia, Blisters, Blue red color, Breaks in skin, Tissue ulceration, Infection sets in and then the tissue dies
Risk Factors:
- Immobility
- Contractures
- Decreased nutrition
- Anemia
- Obesity
- Sepsis
Diagnosis:
- Homan's sign is only present in 30% of true cases
- False positive can occur with mm injuries, Achilles tendonitis, ruptured bakers cyst
- 50% of positive tests are not venous thrombosis
- Diagnosis is made on Hx, Doppler ultrasound, venous plexus scanning
Treatment:
- Oral antibiotics are effective
- Debridement and clean hygiene
- Topical antibiotics are not effective
Therapist concerns:
- Position accordingly. Encourage adequate hydration and nutrition
- Reposition every 2 hrs
- No hot or ice hydro
- Inspect skin each Tx and record
- Use moisturizing lotion or cornstarch
- Bolster, pillow to take pressure off
- Maintain current ADLs, activity level, ROM, Mobility
- Lower head of bed for venous return
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